Researchers from Xishuangbanna Tropical Botanical Garden of the Chinese Academy of Sciences and collaborators examined how biotic factors and abiotic factors affect the aboveground biomass at two spatial scales in a tropical seasonal rainforest in southwestern China.
A recent collaborative study has deciphered the cryo-EM structure of the spinach type I PSII-LHCII megacomplex, providing insights into the principles of higher-order assembly and the potential regulatory mechanisms of plant PSII under low-light conditions.
A research team led by Eric H. Xu and WU Canrong from the Shanghai Institute of Materia Medica presented cryo-electron microscopy structures of apo DP2-Gi complex, a DP2-Gi complex bound to endogenous ligand PGD2, and a DP2-Gi complex bound to indomethacin, a ligand known to favor β-arrestin signaling. These structures revealed distinct binding modes and provided critical insights into the receptor's activation and signaling bias, highlighting the role of lipid interactions.
Craniopharyngioma has long been a daunting challenge for neurosurgeons and endocrinologists alike. Chinese researchers recently revealed new insights on the growth of craniopharyngioma and identified a potential therapeutic treatment. Understanding this complex tumor’s growth mechanism and identifying a potentially effective, non-invasive medication to treat it mark a significant step forward.
Recent collaborative research has revealed the specific regulatory mechanism involved in phase separation during plant flowering. This study was led by Professor HOU Xingliang from the South China Botanical Garden of the Chinese Academy of Sciences, and Professor MIAO Yansong from Nanyang Technological University in Singapore.
A research team led by Prof. WANG Haoyi from the Institute of Zoology (IOZ) of the Chinese Academy of Sciences has developed a chimeric antigen receptor T (CAR-T) cell exhaustion model and a functional screening platform for identifying compounds that can rejuvenate exhausted T cells. Using this innovative platform, the team identified the small-molecule compound miltefosine, which significantly enhances the tumor-killing activity of CAR-T cells.
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